Bone regeneration research has taken a significant leap forward with the discovery of a crucial mechanism that could transform treatments for bone disorders. Scientists have identified how Discoidin Domain Receptor 2 (DDR2) enhances Bone Morphogenetic Protein (BMP)-dependent bone regeneration while mitigating the risk of heterotopic ossification (HO), offering promising therapeutic opportunities. This breakthrough sheds light on how DDR2 regulates BMP activity, paving the way for safer and more effective interventions in bone repair and related conditions.
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